Cell Aging Pathways
David explains how mTOR, IGF One, and autophagy pathways interact to influence cell aging. Lifestyle choices can impact these pathways, affecting the cell's ability to maintain epigenetic information and stay youthful.In this clip
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Related Questions
I'm concerned about the role of mTOR and IGF-1 in tumor growth as discussed in the episode 19. Can we live forever? A conversation about aging with David Sinclair PhD and the clip mTOR and Insulin Insights from the Dr. David Sinclair: The Biology of Slowing & Reversing Aging | Huberman Lab Podcast #52. Can we achieve a balance between activating these pathways for muscle growth and strength training, and downregulating them through fasting? For example, could a cycle of 1 month of muscle-building weight training followed by a week of fasting help strike that balance? What might an optimal approach look like?
I'm concerned about the role of mTOR and IGF-1 in tumor growth as discussed in the episode 19 of the Huberman Lab Podcast, "Can we live forever? A conversation about aging with David Sinclair PhD," and the clip "Diet and Longevity." Is there a balance that can be achieved between activating these pathways for muscle growth and strength training, and downregulating them through fasting? For example, could a cycle of 1 month of muscle-building weight training followed by a week of fasting help strike that balance? What might an optimal approach look like?
I'm concerned about the role of mTOR and IGF-1 in tumor growth as discussed in the episode 19. Can we live forever? A conversation about aging with David Sinclair PhD and the clip Diet and Longevity. Is there a balance that can be achieved between activating these pathways for muscle growth and strength training, and downregulating them through fasting? For example, could a cycle of 1 month of muscle-building weight training followed by a week of fasting help strike that balance? What might an optimal approach look like?